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20090111/DC1). Introduction {#ddc31226-sec-0010} ============ Osteoarthritis (OA) has a multifactorial etiology that involves: pain, inflammation, and functional disability.[1](#ddc31226-bib-0001){ref-type=”ref”} Although genetic variation in the inflammatory response to different substrates has been shown to contribute to the pathogenesis of OA, the ultimate risk of developing OA is still unknown. Pathology studies suggest that OA may be characterized by a progressive range of pathologies, including primary interoarthritis, interlapsular synovitis, and arthritis and either surgical or biologic therapy.[2](#ddc31226-bib-0002){ref-type=”ref”} In addition, the majority of OA cases are surgically managed with arthrolysis. Although the outcomes of joint replacement surgery are significantly different among these groups, certain clinical and preclinical data warrant careful consideration when treating joint replacement arthritis; however, these limitations can be overcome by incorporating evidence‐based and validated techniques. Several case reports of clinically relevant OA have become available to date, including the recently published Report of A T‐PALP Study, the largest study to describe OA after PALP in a large Dutch group of knee OA patients. The results appear to be in line with the objectives of this research ([Allan *et al,* [2014](#ddc31226-bib-0007){ref-type=”ref”}). PALP, a newly developed type of arthroscopic approach for arthroplasty,